By W. Finn, L. Peterson
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Br J Haematol 1989;72:294-295. Myelodysplastic/Myeloproliferative Diseases 43 65. , Pierre R. Myelodysplastic/myeloproliferative disease, unclassifiable, in Jaffe ES, Harris NL, Stein H, Vardiman JW (Eds): World Health Organization Classification of Tumours. Pathology and Genetics of Tumours of Haematopoietic and Lymphoid Tissues. Lyon: IARC Press, 2001. Chapter 3 RELEVANCE OF PATHOLOGIC CLASSIFICATIONS AND DIAGNOSIS OF ACUTE MYELOID LEUKEMIA TO CLINICAL TRIALS AND CLINICAL PRACTICE Martin S. Tallman Northwestern University Feinberg School of Medicine, and Robert H.
3 to nearly but are usually normal in number. 49 Immature neutrophils (promyelocytes, myelocytes) usually account for fewer than 10% of the WBCs. Mild basophilia is sometimes present, but basophils usually account for fewer than 2% of the WBCs. 4 Although a number of such cases are associated with translocations that involve PDGFRbeta on chromosome 5,12-14 in most cases of CMML with eosinophilia a specific genetic defect has not yet been identified (Figure 2-5). 2, 52 Granulocytes are often the most prominent bone marrow component, but an increase in erythroid precursors may be seen as well (Figure 2-6).
2 Cytogenetic/Genetic Findings No specific cytogenetic or genetic abnormalities have yet been identified in CMML. Abnormal karyotypes are reported in 20-40% of cases of CMML. 12-14 Although important as a model for leukemogenesis, this translocation, as well as others involving the PDGFRbeta on chromosome 5, are uncommon. Abnormalities involving MLL at 11q23 are also unusual in CMML, and, if present, suggest a diagnosis of AML rather than CMML. 1 Ph+, BCR/ABL+ CML The distinction between BCR/ABL+ CML and CMML is usually made on the basis of morphology combined with cytogenetics and molecular genetic studies.
Hematopathology in Oncology by W. Finn, L. Peterson